hello Go granny
I am also going through low blood sugars and what the other guy said about drinking soda sheesh .. What all of you need to know here is that most times the cells do not stop growing in your pancreas,it has taken a year to go through all the tests I have had and the last one the calcium stimulation test showed that it has moved up into the upper regions of the pancreas and I am sched for partial removal and spleen removal.. there are meds for this it is called Precose or generic is acarbose. it does work for a while until the growth of the cells and the meds can not slow down the release well enough..what the guy with the coke syrup doesnt realize is that any food that is carb related sets off the cycle.. Look up
Nesidioblastosis and NIHPS I will post what I have here for you to look up.. Do not believe that anything can cure this even reversal of your RNY isnt a deff help.. I have come across to much info that shows different.. and you need to see a endo in a place like the Mayo or like me Jefferson the lower hospitals do not have enough info to help.. most docs dont even know what your talking about. you can die from the low blood sugars.. as you are well aware.. and even like me the removal of most of my pancreas isnt a sure thing if the cells keep growing I will have to go back.. The doctor I am seeing is versed in this problem and has done work on it with several patients. No matter what I eat anymore I cant get my sugar to break 200 and I can drop thirty points in fifteen min. Any thing that you do in the am to start your system like any type of exercise will start the cycle as well I have found. You will have to go through cts and Mri and have them run what is called a mixed meal study the blood usually has to go to the mayo to be tested.. this problem is early on and you need to see ppl who know what they are doing and not guessing.. here are some links for you and I hope all goes well.. my surgery is aug 23, I am not happy about it but I can tell its getting worse and this may keep me from total loss of my pancreas but considering the fourth artery tested in the calcium stimulation test showed some raised levels I dont have much hope in keeping the rest of my pancreas in a few years...
NIPHS
Mayo Clinic physicians have recognized and reported on a seemingly rare but serious complication following gastric bypass called NIPHS (non-insulinoma pancreatogenous hypoglycemia syndrome) or post-bariatric surgery hypoglycemia. After a person eats, this condition can result in very low blood sugar levels that lead to severe neurologic symptoms, including visual disturbances, confusion and (rarely) seizures.
Mayo physicians in Minnesota have evaluated and treated several patients with NIPHS. When medical and diet therapies do not work, surgical removal of part of the pancreas has resulted in marked improvement of symptoms for most patients. If the symptoms described above occur, patients should notify their physician immediately. Until this condition is controlled, patients should avoid driving motorized vehicles or performing tasks that could affect the safety of those around them.
Nesidioblastosis
http://en.wikipedia.org/wiki/Nesidioblasto
sis
Nesidioblastosis is a controversial medical term for hyperinsulinemic hypoglycemia attributed to excessive function of pancreatic beta cells with an abnormal microscopic appearance. The term was coined in the first half of the 20th century. The abnormal histologic aspects of the tissue included the presence of islet cell enlargement, islet cell dysplasia, beta cells budding from ductal epithelium, and islets in apposition to ducts.
By the 1970s, nesidioblastosis was primarily used to describe the pancreatic dysfunction associated with persistent congenital hyperinsulinism and in most cases from the 1970s until the 1980s, it was used as a synonym for what is now referred to as congenital hyperinsulinism. Most congenital hyperinsulinism is caused by different mechanisms than excessive proliferation of beta cells in a fetal pattern and the term fell into disfavor after it was recognized in the late 1980s that the characteristic tissue features were sometimes seen in pancreatic tissue from normal infants and even adults, and is not consistently associated with hyperinsulinemic hypoglycemia.
However, the term has been resurrected in recent years to describe a form of acquired hyperinsulinism with beta cell hyperplasia found in adults, especially after gastrointestinal surgery.[1][2][3]
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