New research indicates that preventing cancer cell growth can be as simple as eating the right foods. Improve your nutrition know-how. Fill up on the following foods that starve cancer. Added to Articles on Fri 09/10/2010 Disturbing new research suggests that microscopic cancer, small cancer cells that can only been seen under a microscope, is widely prevalent. A recent study of women in their 40s indicated that 40% of them had microscopic breast cancer. Even more shocking, almost 100% of people in their 70s will have microscopic cancer in their thyroid glands.
A microscopic tumor can grow up to 16,000 times its original size in as little as 2 weeks. But new groundbreaking research from The Angiogenesis Foundation proposes that you can stop cancer before it begins to grow. This new preventive approach is called anti-angiogenesis. To learn more about anti-angiogenesis and the groundbreaking research at The Angiogenesis Foundation, click here. Anti-angiogenesis encourages that, by changing the way you eat, you can change your internal environment, thereby depriving cancer cells the opportunity to grow and multiply.
Certain foods, eaten in the correct portions and frequency, can provide cancer-starving benefits. Below are 5 foods to eat that can prevent cancer growth:
1.Bok Choy This type of Chinese cabbage contains brassinin; a powerful cancer-fighter, also found in broccoli, cauliflower, kale and Brussels sprouts. Bok Choy should be eaten 3 times a week, in 1/2 cup servings to obtain its full benefits.
2.Cooked Tomatoes have more cancer-fighting properties than raw tomatoes. Both contain the molecule lycopene, but heating the tomato changes its chemical structure and makes the benefits more readily available to your body. You should eat 2-3 (1/2 cup) servings of cooked tomatoes a week.
3.Flounder This fish is rich in omega-3 fatty acids and low in mercury. Three 6-ounce servings a week is ideal.
4.Strawberries The antioxidants in this berry help fight cancers. You should eat 1 cup a day, including the juice.
5.Artichokes contain 3 different cancer-fighting molecules. Enjoy Â¼ cup of hearts per day.
broccoli/broccoli sprouts, inhibits breast cancer stem cells.
Sulforaphane, a dietary component of broccoli/broccoli sprouts, inhibits breast cancer stem cells.
Department of Pharmaceutical Sciences, College of Pharmacy, University of Michigan, Ann Arbor, MI 48109, USA.
The existence of cancer stem cells (CSCs) in breast cancer has profound implications for cancer prevention. In this study, we evaluated sulforaphane, a natural compound derived from broccoli/broccoli sprouts, for its efficacy to inhibit breast CSCs and its potential mechanism.
Aldefluor assay and mammosphere formation assay were used to evaluate the effect of sulforaphane on breast CSCs in vitro. A nonobese diabetic/severe combined immunodeficient xenograft model was used to determine whether sulforaphane could target breast CSCs in vivo, as assessed by Aldefluor assay, and tumor growth upon cell reimplantation in secondary mice. The potential mechanism was investigated using Western blotting analysis and beta-catenin reporter assay.
Sulforaphane (1-5 micromol/L) decreased aldehyde dehydrogenase-positive cell population by 65% to 80% in human breast cancer cells (P < 0.01) and reduced the size and number of primary mammospheres by 8- to 125-fold and 45% to 75% (P < 0.01), respectively. Daily injection with 50 mg/kg sulforaphane for 2 weeks reduced aldehyde dehydrogenase-positive cells by >50% in nonobese diabetic/severe combined immunodeficient xenograft tumors (P = 0.003). Sulforaphane eliminated breast CSCs in vivo, thereby abrogating tumor growth after the reimplantation of primary tumor cells into the secondary mice (P < 0.01). Western blotting analysis and beta-catenin reporter assay showed that sulforaphane downregulated the Wnt/beta-catenin self-renewal pathway.
Sulforaphane inhibits breast CSCs and downregulates the Wnt/beta-catenin self-renewal pathway. These findings support the use of sulforaphane for the chemoprevention of breast cancer stem cells and warrant further clinical evaluation.
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