When reactive hypoglycemia was discovered it was called "disglycemia" or in other words unstable blood sugar. Reactive Hypoglycemia was discovered when Insulin for diabetics was invented. Diabetic people who used too much insulin had terrible symptoms (spaciness, dizzyness, anxiety, sweating, fainting, aggressivity, blurred vision and speech, headache, panick, nausea and more) that were similar to symptoms observed of non-diabetic people which lamented fatigue and confusion and who appeared to be otherwise healthy.
This demonstrated that these people had an excessive production of insulin. This excessive production of insulin is seen on people with insulin resistance and glucose intolerance. Your cells don't admit the sugar and 10 times more insulin is secreted by your pancreas to counteract the cellular resistance. This amount of excessive insulin brings your blood sugar down. Adrenalin is then released to bring sugar up. Adrenalin is what causes the anxiety, fear, panick, phobia, irritability, aggressivity, sweating episodes.
Since the blood glucose metabolism is made to maintain balanced blood sugar, unstable blood sugar is the expression of an impaired blood glucose metabolism just like diabetes is. Even when a normal person eats something sugar their blood glucose never spike above 150. Then one hour later it is 130, then two hours later it is 110 and eventually three or four hour later it is back to fasting levels (80 or 90)
A person with reactive hypoglycemia eats carbs, the blood sugar spike even above 200. After an hour it is still high. This is when the pancreas releases a lot of insulin. Suddenly two hours or more later (after the excessive insulin has done its job) it quickly and suddenly plummet to 40 or even 30 where anything below 70 is hypoglycemia.
The graph looks like high stylized mountains or roller coaster. The graph of a normal person looks like a flat line with a small hill curve at the beginning.
Notice that the initial response to the carbs the person ate (blood sugar spiking above 200 and being still high after 1 hour) is exactly the same exact thing that happens in diabetes.
But in diabetes the pancreas can't produce the insulin so the sugar is still high many hours later. In Reactive Hypoglycemia (or I should say at this point pre-diabetes) the pancreas is still able to produce enough insulin, actually too much of it.
That's why the discovered of Reactive Hypoglycemia said that the low blood sugar of today is the diabetes of tomorrow. Not only hypoglycemia is basically diabetes without the insulin shortage, but a pancreas which many times a day produce 10 times for insulin, eventually will stop producing insulin.
It has been known for many years that mostly young people (like us) have Reactive Hypoglycemia because as people reached their 40's hypoglycemia "goes away". Unfortunately this is not because they're getting better, but because the body no longer can produce enough insulin to cope with the high blood sugar. So at this point they have fasting hyperglycemia and probably are diagnosed with diabetes. I know a lot of people who was diagnosed with reactive hypoglycemia (which they never managed properly) and eventually became diabetics. Most diabetics suffered from pre-diabetes and unstable blood sugar symptoms, the same you and I suffer from.
Since your pancreas secretions are not impaired yet you won't sign an evidence of the big mess that happens when you eat in a fasting test. The only way to see what really happens (sugar going up, then down, excessive insulin being released, adrenalin surge) is to see what happens in your blood after you've eaten carbs. This test is called Glucose Tolerace Test and check what happens in your blood every 30 minutes after you've drunk a glucose drink.