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Certain HIV positive, but negative tests (Page 65)

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March 3rd, 2013
Dosent anyone post on here no more ??
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replied March 3rd, 2013
Dosent anyone post no more ? ?
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replied March 3rd, 2013
Dosent anyone post no more ? ?
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replied March 3rd, 2013
This forum is new too me my symptoms are NOT !!
6 months of hell still no answers !!




20/1/13


Ongoing symptoms to date

Pain down spine seems tight
hairy tongue
Blurred vision
Sore roof of mouth and soft palette
Body legs hips pains
Red eyes
Dark urine
Swollen lymph nodes


Tests performed outside of window

Hiv 1.
11 x p24 antigen tests
11 x antibody tests
1 x guantitive pcr rna test


Hiv 2
11 x p24 antigen tests
11 x antibody tests
1 x guantitive pcr rna test


Htlv
antibody/antigen post window period @ (17 weeks) cdc guidelines say window period is 45-90 days max. I tested 120 days


Hep A hep B hep C -

Antibody and quantitive pcr test and antibody/antigen for hep B

Mrsa Pcr swab x 3

Toxoplasmosis test



PCR common viral pathology test

Norovirus
Cytomegalo virus
Epsten barr virus
Infuenza a b c
Rhinovirus

Further tests immune test @ ten weeks

Cd4 count 1000 ( normal/good range )

Cd3 slightly elevated ( sign of infection paired with generalised swollen lymph glands )

Cd8 within normal range

Further tests

Eye examination
Oral examination
Ear examination
Full body examination
Reflex test
Head x ray
Chest x ray
Celiac disease antibody test
Thyroid function
Blood sugars. (diabetes)
Full blood count x4
Calcium quantity
Ferret-in levels
Anaemia test
Esr inflammation test
Kidney function
Liver function
Bacteriology on urine
Immune response test


Just like all CFS patients are different. For some CFS patients, CFS is caused by HHV6. For others, it's mycoplasma. For others, it's bacillus, squalane, mercury, depleted uranium, benzene, or chemtrails; Or, for others, it's a combination of pathogen, environment, oxidative stress, and genetic predisposition.


Still no answer too what is wrong i cant help thinking the worst because of the way i feel !
Im certain i am infected with some type of viral pathogen blood born or not.
Im scared for my future.
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Users who thank dontgiveup85 for this post: TonyDewitt 

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replied March 3rd, 2013
Extremely eHealthy
DG,

The symptoms you listed are the same ones I am suffering from, and I believe that the cause is HTLV.

Best wishes.
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replied March 5th, 2013
Extremely eHealthy
Today I received the following mail from the UK HTLV team, where they once again defend HTLV testing and lack of treatment:

From: HTLV
Date: Mar 5, 2013 4:57:54 AM
Subject: RE: RE: Testing and treatment of HTLV


Dear patient,



Thank you for your reply. We do not agree that HTLV-I is extremely difficult to detect nor that it elicits a weak or non-existent immune response.

High antibody titres to HTLV-I are characteristic feature of the infection allowing pooling of blood samples for testing without loss of sensitivity.



Tofacitinib would be interesting to examine but is not part of any current studies.



Best Wishes

HTLV Team
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Users who thank TonyDewitt for this post: Definatelyworried 

replied March 6th, 2013
Experienced User
What happend to everyone? ????????
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Users who thank Depressedman for this post: TonyDewitt 

replied March 6th, 2013
Experienced User
What happend to everyone? ????????
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Users who thank Depressedman for this post: TonyDewitt 

replied March 17th, 2013
Experienced User
Hi How is everyone doing, i still have all my symptoms going on.
I did a recent HTLV Western blot test with Biomanis Lab in France, the test came negative for all core protines of HTLV virus.Gag P19 I/II
Gag p24 I/II
Env gp46 I/II
Env gp21 I/II
Gag p 19 I
Env gp 46 I
Env gp46 II
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Users who thank adviseme for this post: TonyDewitt 

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replied March 17th, 2013
Extremely eHealthy
AdviseMe,

Thanks for doing the HTLV Western Blot test - this is what I had asked everyone to get done, so I am glad that you did it and posted the results for HTLV's individual viral proteins. The fact that NONE of the several proteins that you listed tested positive makes this disease even more mysterious.

Everyone else,

Before the traffic stopped on this forum due to the attacks, I had asked everyone else to also get the Western Blot for HTLV test like AdviseMe had done. Some of you had volunteered to get this test done, but never reported the results to the forum. Since the symptoms of HTLV match our disease (see DGU85's list of symptoms above), we all need to get tested in this manner, at least to prove as a group that the testing is consistent.

Best wishes.
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replied March 19th, 2013
Experienced User
Ustas? How are you after the Mycoplasma treatment? Are you back to normal? I wanna test for Mycoplasma. What kind of Doctor can teat me for it? Plz reply..

Symptoms still today after exposure. 2/19/12

Something in throat. Like hair istuck or bone feeling.
Very itchy skin. Red bumps on body. Follucollitis
Leg joints and muscle pain
Marks on lower stomach. Look like strech marks. ?
Congested nasal and throat
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Users who thank Depressedman for this post: TonyDewitt  TonyDewitt 

replied March 19th, 2013
Experienced User
Depressedman

Things are the same on my part. Nothing seems to be helping.

I'm still experiencing itchy skin and burning skin sensation in legs.

All the best guys.


I DID NOT APPRECIATE THE ARGUMENT/DISPUTE THAT CAUSED A BREAK IN THE POST ON THIS FORUM.

ARE WE HERE TO HELP EACH OTHER?
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Users who thank Ustas for this post: TonyDewitt 

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replied March 19th, 2013
Extremely eHealthy
Ustas,

Yes, we are here to help each other.

Best wishes.
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replied March 26th, 2013
Extremely eHealthy
In the vein of helping each other, I want to tell you that I began taking a new supplement a few days ago that has made me feel better (i.e. reduced pain and stiffness) - it's 5 dollars a bottle on the Swanson Vitamin site:

Swanson Premium Full-Spectrum Chinese Skullcap
Caps 90 400 mg Caps

For 5 dollars a bottle, I think it's worth trying. The main ingredient (Scutellaria baicalensis) has been shown to have antiviral activity, especially with respect to HTLV. Here's an excerpt from a medical paper:

Baicalin inhibited reverse transcriptase activity in HTLV-I—infected cells as well as the activity of purified reverse transcriptase from Moloney murine leukemia virus and Rous-associated virus type 2. These results suggest that baicalin may be a potential therapeutic agent against HTLV-I-associated T cell diseases. (I am taking ONE tablet a day).

Best wishes.
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replied March 27th, 2013
Experienced User
I'm so exausted looking for answers.. what's bugging me the most these days is my skin.. its just so ugly. Itchy and red bumps that later leave dark spots.. I feel so nasty and infected.. bumps on arms legs chest and face.. not to the extreme but I notice new onew ones everyday. I just don't know what to do.
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Users who thank Depressedman for this post: TonyDewitt 

replied April 28th, 2013
Experienced User
same itchy \ Bumby Skin here Dman,
still don't know what to do , ??

how is your test results going on man ..
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replied March 28th, 2013
Extremely eHealthy
There are no approved therapeutics for the most deadly nonsegmented negative-strand (NNS) RNA viruses, including Ebola (EBOV). To identify chemical scaffolds for the development of broad-spectrum antivirals, we undertook a prototype-based lead identification screen. Using the prototype NNS virus, vesicular stomatitis virus (VSV), multiple inhibitory compounds were identified. Three compounds were investigated for broad-spectrum activity and inhibited EBOV infection. The most potent, CMLDBU3402, was selected for further study. CMLDBU3402 did not show significant activity against segmented negative-strand RNA viruses, suggesting proscribed broad-spectrum activity. Mechanistic analysis indicated that CMLDBU3402 blocked VSV viral RNA synthesis and inhibited EBOV RNA transcription, demonstrating a consistent mechanism of action against genetically distinct viruses. The identification of this chemical backbone as a broad-spectrum inhibitor of viral RNA synthesis offers significant potential for the development of new therapies for highly pathogenic viruses.
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replied April 1st, 2013
hiv ab test
I have the same problem . i tested negative after 3 month. 3.5 months.and 5.5 month . but I have submandibular lymph node and chronic diarrhea that started 9 month after exposure and lasted for 1 month. please help me .I just want to know if hiv ab(1,2) after 5.5 month conclusive?
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replied April 2nd, 2013
Extremely eHealthy
Yes, testing negative for HIV at three months (and beyond) conclusively proves that you are HIV negative. At this point you need to look for other causes (HTLV, Hepatitis, etc).

Best wishes.
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replied May 31st, 2013
Hi myassef;

I'm curious about your submandibular lymph node, have you had it checked already ? MRI, CAT SCAN, etc ?

how about the diarrhea ? do you still have it ?

These are most bothersome symptoms I feel also. Out of curiosity, do you have sebum in the forehead & nose ?
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replied April 6th, 2013
Experienced User
Elimination of the leader. Induced cancer?

Antonio Brizuela Paneque *

The suspicion that President Hugo Chavez died in March due to a cancer caused actualizes the old craft eliminate the leaders anywhere in the world through what we might call "high-tech crime."

With the reappearance of news about the existence of scientific methods to kill, began a debate in Caracas after the announcement of a thorough investigation to determine whether the tumor was sent who killed their leader.

The possibility that it may have been induced, in a planned way, this type of disease monopolizes the scientific and political moment, given the statements that link on the front and not well-known studies on the subject, particularly in the United States.

Although the distrust with respect to these practices in that country, are not a new thing, shooting force complaints, such as those of Julian Assange, who has referred to certain U.S. scientific experiments at a place called Fuerte Detrick.

Assange, known for the dissemination of documents that compromise through
WikiLeaks said in March 2012 that this special section of the Virus Department of the Center for Biological Research is used since 1975 for this type of experiments.

The place is known as "Fredrick Installations for Research of Cancer" and depends on the Department of Defense, the CIA and the National Institute of Cancer, assured Assange in an interview with Globo News.

MADE IN LABORATORY

According to this research ultrasegrete cited by the source, you can create programs on cancer virus extremely aggressive, lethal and also immune. Its scientific name is Virus Human T-cell Leukemia (HTLV).

Through these programs, have created an artificial highly invasive malignant cells are able to propagate into the body through the blood or lymph, to destroy any defense and cause incurable metastases.

It is, said in another way, of an alteration in the genetic material of human cells to induce tumor by inoculations that may use different roads, according to the journalist Guatemalan Percy Alvarado Godoy.

Among the resources of these laboratories that work for more than 40 years is that of stem cells (stem cells), treated and monitored by mutations to make them more diverse and malignant phenotypes of rapid development, according to the source.

The U.S. agency The Associated Press, for its part, published in 2007 an article based on declassified documents considered "one of the most enduring secrets of the Cold War."

"The United States Army has studied," said in this regard the article, "the possibility of using radioactive poisons to assassinate important people, military or civilian."

HOW MANY OFFICERS WILL BE DEAD IN THIS WAY?

Even President Chavez has done in Venezuela a kind of premonition, being the first that the declarations of December 28, spoke about the possible diseases induced, when he referred to contemporary getting several Latin American presidents.

The possible liability of Washington was suggested by the late president when he referred to the appearance of cancer in Brazil Dilma Rousseff and Lula da Silva, Argentina's Cristina Fernandez, Fernando Lugo paraguaio and himself.

"It would not be strange that they had created a technology to induce cancer and no one knows so far," said Chavez, then without knowing some details on the issue that are now handled by those who ordered the search.

The questions of the leader, the answers seem to indicate that he himself was a victim can be expanded without limit in time and space.

How many leaders, revolutionaries, progressives or people who do not agree to any government or authority have been, throughout history, victims of crimes based on these formulas to build inhuman death?
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replied April 8th, 2013
Extremely eHealthy
I came across something called HRES (HTLV Related Endogenous Sequence). Since our symptoms seem to be HTLV related, but we are testing negative for HTLV, perhaps this is the cause of our symptoms?

Disease relevance of HRES1
The HRES-1/1 genomic locus is transcriptionally active in lymphoid cells, melanoma cells, and embryonic tissues [1].
Phylogenetic analysis suggests that HRES-1/1 entered the genome in primates, presumably as an exogenous retrovirus [1].
From the deduced amino acid sequence of HRES-1/1 p25, residues 6-36 show a sequence homology of 32% and 39% to gag region segments of HTLV-I and HTLV-II, while residues 104-139 display a sequence homology of 33% and 28% to the gag regions of human immunodeficiency virus type 2 (HIV-2) and feline sarcoma virus (FSV), respectively [1].
Molecular analysis of endogenous retrovirus HRES-1: identification of frameshift mutations in region encoding putative 28-kDa autoantigen [2].
A possible involvement of HTLV-1-related endogenous sequence 1 (HRES-1) in autoimmune diseases has been recently reported [2].
High impact information on HRES1
The presence of a human T-cell lymphotropic virus (HTLV)-related endogenous sequence, HRES-1, in the human genome has been documented [3].
Human T-cell lymphotropic virus (HTLV)-related endogenous sequence, HRES-1, encodes a 28-kDa protein: a possible autoantigen for HTLV-I gag-reactive autoantibodies [3].
The HRES-1 genomic locus is transcriptionally active and contains open reading frames [3].
Thus, HRES-1 is a human endogenous retroviral sequence capable of protein expression [3].
The data indicate that HRES-1/p28 may serve as an autoantigen eliciting autoantibodies cross-reactive with HTLV-I gag antigens [3].
Biological context of HRES1
The human T-cell leukemia virus-related endogenous sequence (HRES1) is located on chromosome 1 at q42 [4].
By nucleotide sequence analysis, HRES-1/1 contains two potential open reading frames capable of encoding a p25 and a p15 [1].
Human T-cell leukemia virus (HTLV) type I-related endogenous sequences (HRES) have been cloned from a human genomic library [1].
Infection by HIV elicited a coordinate down-regulation of CD4 and up-regulation of HRES-1/Rab4 in PBL [5].
Family studies showed that Hin dIII genotypes of the HRES-1 locus are inherited in a Mendelian pattern [6].
Anatomical context of HRES1
The expression of HRES-1 salivary gland may explain its antigenicity in a small proportion of Sjgren's syndrome patients as well as suggesting mechanisms whereby it may contribute to the chronic inflammation of autoimmune disease [7].
Associations of HRES1 with chemical compounds
Antigenic epitopes of HRES-1 and the retroviral gag-related region of the 70-kd protein component of U1 small nuclear RNP, which share 3 consecutive highly charged amino acids (Arg-Arg-Glu), an additional Arg, and functionally similar Arg/Lys residues, represent cross-reactive epitopes between the two proteins [8].
Other interactions of HRES1
However antisera generated against selected HRES-1 peptides failed to detect a 28-kDa protein deduced from the translated gag ORF and described previously [2].
Using an enzymatic amplification technique, we found expression of the endogenous retroviral sequences, HRES-1, HERV-K10 and ERV3 in most samples of peripheral blood mononuclear cells from MS patients and controls without obvious differences between these two groups [9].
Antibody reactivity to the HRES-1 endogenous retroviral element identifies a subset of patients with systemic lupus erythematosus and overlap syndromes. Correlation with antinuclear antibodies and HLA class II alleles [8].
In the present study we searched for an association between the human endogenous retroviral element HRES-1 and multiple sclerosis (MS) [10].
Analytical, diagnostic and therapeutic context of HRES1
HRES-1/p28 Western blot reactivity was observed in 12/23 TTV PCR-negative donors and 43/58 TTV PCR-positive donors (P < 0.0281) [11].
Expression of HRES-1, detected by antibodies and Northern blots, was found in lymphoblastoid cells, salivary gland biopsy sections and salivary gland epithelial cells in culture [7].
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replied April 8th, 2013
Extremely eHealthy
Human T-cell leukemia virus (HTLV) type I-related endogenous sequences (HRES) have been cloned from a human genomic library. HRES-1/1 is present in DNA of all normal donors examined. By nucleotide sequence analysis, HRES-1/1 contains two potential open reading frames capable of encoding a p25 and a p15. A 684 bp flanking region 5' from the first ATG codon of p25 contains a TATA-box, a poly-adenylation signal, a putative tRNA primer binding site, and inverted repeats at locations which are typical of a retroviral long terminal repeat. Phylogenetic analysis suggests that HRES-1/1 entered the genome in primates, presumably as an exogenous retrovirus. From the deduced amino acid sequence of HRES-1/1 p25, residues 6-36 show a sequence homology of 32% and 39% to gag region segments of HTLV-I and HTLV-II, while residues 104-139 display a sequence homology of 33% and 28% to the gag regions of human immunodeficiency virus type 2 (HIV-2) and feline sarcoma virus (FSV), respectively. This suggests that the original exogenous virus infecting primate may be chimeric in structure. The HRES-1/1 genomic locus is transcriptionally active in lymphoid cells, melanoma cells, and embryonic tissues.
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replied April 8th, 2013
Experienced User
Defective human T-cell lymphotropic virus type I (HTLV-I) provirus in seronegative tropical spastic paraparesis/HTLV-I-associated myelopathy (TSP/HAM) patients.

Infection with human T-cell lymphotropic virus type I (HTLV-I) have been associated with the development of the tropical spastic paraparesis/HTLV-I-associated myelopathy (TSP/HAM). We studied the presence of HTLV-I provirus in peripheral blood mononuclear cells (PBMC) from 72 Chilean patients with progressive spastic paraparesis by polymerase chain reaction: 32 seropositive and 40 seronegative cases. We amplified different genomic regions of HTLV-I using primers of 5' ltr, tax, env/tax, pX, pol and env genes. These genes were detected from all seropositive patients. The seronegative patients were negative with 5' ltr, pol, env, and pX primers. However, amplified product of tax and env/tax genes was detected from 16 and four seronegative patients, respectively. Three of them were positive with both genetic regions. The results of this study show that the complete HTLV-I provirus is found in 100% of seropositive cases. In seronegative cases, clinically very similar of seropositive cases, was found only tax gene in 42.5% (17/40) of patients. These results suggest the presence of a defective HTLV-I provirus in some seronegative patients with progressive spastic paraparesis, and suggest a pathogenic role of this truncate provirus for a group of TSP/HAM.
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Users who thank adviseme for this post: TonyDewitt  Definatelyworried 

replied April 9th, 2013
Experienced User
Hello, I am back. I moved over to the Phoenix rising group. I thought everyone had left.

Anyway. I tried to get the tests Tony suggested but my dr. refused

I fluctuate between 80 and 90% - I actually worked out the other day.

I need to re engage and find another dr. I tried the center out west but they were less than helpful.

At this point my symptoms have changed a little.

Tiredness, headache, some confusion, rapid heart, chest an stomach discomfort but not pain, red eyes, loss of appetite and now weird ibs symptoms.

For me the original infection is seems less but the overarching issues appear to be set. I have not seen any additional improvement.

I am searching for a new dr. to start over with.
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Users who thank wyomingscared for this post: TonyDewitt  Definatelyworried 

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replied April 9th, 2013
Extremely eHealthy
WS,

Same here - less pain, but same symptoms that you mentioned, especially with the red eyes & IBS symptoms. I eat a cup of yogurt for breakfast to deal with the IBS problems, which I believe are caused by Candida / yeast infection, which yogurt combats. Thanks for trying to get tested, and seeing the reluctance of doctors to not only refuse to test, but refuse to deal with this disease.

Best wishes.
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replied April 9th, 2013
Extremely eHealthy
Someone in this forum wrote me that during a phone call Doctor Poeisz in upstate NY made a statement that virtually everyone will test positive for HTLV in two years with antibody testing, and one year with PCR testing. Having this type of statement from a medical professional is powerful to me because it refutes the general belief that every virus can be detected via antibody or PCR within a few months of infection. Applying this rule to my own situation, this means that my 4.5 month PCR test was useless, along with the EIGHT antibody tests that I had in the last 18 months. If Doctor Poeisz truly stands behind this schedule, he should publish this so that other doctors and government health officials can recognize the incubation and window period for this disease.

Best wishes.
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replied April 9th, 2013
Extremely eHealthy
In vitro, four styrylquinoline compounds and two diketo acid compounds significantly inhibited HTLV-1 integration in a dose-dependent manner. All compounds active in vitro decreased cell proliferation ex vivo, although at low concentrations; they also dramatically decreased both normalized proviral loads and the number of integration events during experimental ex vivo primary infection. Accordingly, diketo acids and styrylquinolines are the first drugs that produce a specific negative effect on HTLV-1 replication in vitro and ex vivo, suggesting their potential efficiency for the prevention and treatment of HTLV-1-associated diseases.
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